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Molecule Parameter List for tot_autonomous_CaMKII

The statistics table lists the distribution of a molecule acting either as a substrate, product, enzyme or as a molecule within the network.
The text color of a molecule is highlighted by color.
Statistics
tot_autonomous_CaMKII participated asMoleculeSum total ofEnzymeSubstrate of an enzymeProduct of an enzymeSubstrate in ReactionProduct in Reaction
No. of occurrences1120000

Accession and Pathway Details
Accession NameAccession No.Accession TypePathway Link
CaMKII_200349Network
Shared_Object_CaMKII_2003 CaMKII CaM 
PP1 PP2B 
Model of regulation of CaMKII by Calcium, including parallel excitatory input from CaM and inhibitory input from PP1 as regulated by Calcineurin and PKA. Cell type: neuronal.
Bhalla US. Biophys J. 2004 Aug;87(2):733-44.

tot_autonomous_CaMKII acting as a Molecule in  
CaMKII_2003 Network
NameAccession NamePathway NameInitial Conc.
(uM)
Volume
(fL)
Buffered
tot_autonomous_CaMKIICaMKII_2003
Accession No. : 49
CaMKII
Pathway No. : 202
01000No

tot_autonomous_CaMKII acting as a Summed Molecule in  
CaMKII_2003 Network
Accession NamePathway NameTargetInput
CaMKII_2003
Accession No. : 49
CaMKII
Pathway No. : 202
tot_autonomous_CaMKIICaMKII-thr286
CaMKII***

tot_autonomous_CaMKII acting as an Enzyme in  
CaMKII_2003 Network
 Enzyme Molecule /
Enzyme Activity
Accession NamePathway NameKm (uM)kcat (s^-1)RatioEnzyme TypeReagents
1
  • tot_autonomous_
    CaMKII
     /
    auton_305
  • CaMKII_2003
    Accession No. : 49
    CaMKII
    Pathway No. : 202
    175.00364explicit E-S complexSubstrate
    CaMKII-thr286

    Product
    CaMKII***
        See Hanson and Schulman again, for afterburst rates of phosph.
    2
  • tot_autonomous_
    CaMKII
     /
    auton_286
  • CaMKII_2003
    Accession No. : 49
    CaMKII
    Pathway No. : 202
    174.9970.54explicit E-S complexSubstrate
    CaMKII-CaM

    Product
  • CaMKII-thr286*-C
    aM




  • Database compilation and code copyright (C) 2022, Upinder S. Bhalla and NCBS/TIFR
    This Copyright is applied to ensure that the contents of this database remain freely available. Please see FAQ for details.